✍ Scribed by B. Gato-Rivera; A.M. Semikhatov
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract The rapid, non‐genomic actions of 1,25‐dihydroxyvitamin D~3~ [1,25(OH)~2~D~3~] have been well described, however, the role of the nuclear vitamin D receptor (VDR) in this pathway remains unclear. To address this question, we used VDR(+/+) and VDR(−/−) osteoblasts isolated from wild‐type
## Abstract Interaction between c‐fos and 1,25(OH)~2~ vitamin D~3~ (VD) on the type I collagen synthesis was studied. VD inhibited collagen synthesis and type I collagen mRNA expression in MC3T3‐E1 osteoblastic cells. In contrast, VD reversed the inhibition of collagen synthesis and mRNA expression
## Abstract In chick skeletal muscle cell primary cultures, we previously demonstrated that 1α,25(OH)~2~‐vitamin D~3~ [1α,25(OH)~2~D~3~], the hormonally active form of vitamin D, increases the phosphorylation and activity of the extracellular signal‐regulated mitogen‐activated protein (MAP) kinase