Background. Ascites in patients with hepatocellular carcinoma (HCC) is a poorly characterized subgroup of malignancy-related ascites. Not only the underlying liver disease, but also the tumor growth and spread contributes to the ascites formation. The authors differentiated ascites in HCC from other
Cyclic nucleotides in biological fluids in hepatocellular carcinoma
β Scribed by Geoffrey M. Dusheiko; Joseph Levin; Professor Michael C. Kew
- Publisher
- John Wiley and Sons
- Year
- 1981
- Tongue
- English
- Weight
- 516 KB
- Volume
- 47
- Category
- Article
- ISSN
- 0008-543X
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Abstract The cyclic nucleotides (CN) cAMP and cGMP occur in considerable amounts in body fluids such as urine, blood and cerebrospinal fluid (CSF). Whether changes in the CN concentrations of blood and CSF reflect metabolic events within the nervous tissues is a relevant question both in terms o
The assessment of biological markers as potential indicators of disease aggressiveness is still an open problem in hepatocellular carcinoma. Cell proliferation and extracellular matrix (ECM)-associated antigens and tumor-cell products can be associated with clinical aggressiveness in this tumor type
Alterations in the expression and activity of guanine nucleotide regulatory proteins (G proteins) have been linked to the growth of several human tumors. We hypothesized that the expression and activity of G proteins are altered in human hepatocellular carcinoma (HCC). The expression of Gi and Gs pr
## Abstract Activity of the myelin marker enzyme 2β²,3β²βcyclic nucleotide 3β²βphosphohydrolase (CNP) was assayed in cerebrospinal fluid samples obtained from patients with multiple sclerosis (MS) and other neurological diseases. The enzyme activity was found to be elevated in acute cases of MS and re
A questionnaire-based survey involving 11,801 hemophiliacs from 54 hemophilia centers in the USA and Europe documented the occurrence of hepatocellular carcinoma (HCC) in 10 patients. The crude rate of HCC was 3.2/100,000 patientdyear, at least 30 times higher than the background incidence of this t