𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Correction: Structure of a human insulin peptide–HLA-DQ8 complex and susceptibility to type 1 diabetes

✍ Scribed by Granucci, Francesca; Vizzardelli, Caterina; Pavelka, Norman; Feau, Sonia; Persico, Maria; Virzi, Ettore; Rescigno, Maria; Moro, Giorgio; Ricciardi-Castagnoli, Paola

Book ID
109921660
Publisher
Nature Publishing Group
Year
2001
Tongue
English
Weight
15 KB
Volume
2
Category
Article
ISSN
1529-2908

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Trans-racial studies implicate HLA-DQ as
Trans-racial studies implicate HLA-DQ as a component of genetic susceptibility to Type 1 (insulin-dependent) diabetes
✍ J. Fletcher; C. Mijovic; O. Odugbesan; D. Jenkins; A. R. Bradwell; A. H. Barnett 📂 Article 📅 1988 🏛 Springer 🌐 English ⚖ 948 KB

Type 1 (insulin-dependent) diabetic patients and control subjects of Afro-Caribbean Negroid racial origin were investigated by serological HLA-DR-typing and restriction fragment length polymorphism analysis using DNA probes corresponding to the DQ alpha, DQ beta and DR beta chain genes. Combined ana

The genetic susceptibility to type 1 (in
The genetic susceptibility to type 1 (insulin-dependent) diabetes: Analysis of the HLA-DR association
✍ E. Wolf; K. M. Spencer; A. G. Cudworth 📂 Article 📅 1983 🏛 Springer 🌐 English ⚖ 725 KB

HLA-DR and MT1, MT2, MT3 genotypes have been investigated in 123 Type 1 (insulin-dependent) diabetic subjects and their families. Ninety-eight percent of probands possessed either DR3 (relative risk = 5.0), or DR4 (relative risk = 6.8) or both antigens (relative risk = 14.3), emphasizing the strong

Immunization with an insulin peptide–MHC
Immunization with an insulin peptide–MHC complex to prevent type 1 diabetes of NOD mice
✍ Li Zhang; Brian D. Stadinski; Aaron Michels; John W. Kappler; George S. Eisenbar 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 535 KB

## Abstract ## Background Mutating the insulin B:9–23 peptide prevents diabetes in NOD mice. Thus, the trimolecular complex of I‐A$^{\rm{g7}}$‐insulin B:9–23 peptide‐TCR may be essential for the development of spontaneous diabetes. Pathogenic T cells recognize the B:9–23 peptide presented by I‐A$^