Recent evidence suggests that macrophages and/or other nonparenchymal cells may release important mediators contributing to the hepatic necrosis induced by high doses of acetaminophen (APAP). The nature and causative role of these mediators has remained elusive, however. To investigate the role of t
Compromised anti-tumor responses in tumor necrosis factor-α knockout mice
✍ Scribed by Constantin N. Baxevanis; Ioannis F. Voutsas; Ourania E. Tsitsilonis; Marinos L. Tsiatas; Angelos D. Gritzapis; Michael Papamichail
- Publisher
- John Wiley and Sons
- Year
- 2000
- Tongue
- English
- Weight
- 223 KB
- Volume
- 30
- Category
- Article
- ISSN
- 0014-2980
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