Clinical implications of molecular genetic aberrations in acute myeloid leukemia
✍ Scribed by Sebastian Scholl; Hans-Joerg Fricke; Herbert G. Sayer; Klaus Höffken
- Publisher
- Springer-Verlag
- Year
- 2009
- Tongue
- English
- Weight
- 505 KB
- Volume
- 135
- Category
- Article
- ISSN
- 1432-1335
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## Abstract Hypermethylator phenotype, a propensity of tumors to incur nonrandom concurrent methylation, has been described in several tumors, including acute myeloid leukemia (AML). More recent studies identified methylation of other tumor suppressor genes, DAP‐kinase and SOCS1, singly in AML. We
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