## Abstract Transforming growth factor (TGFβΞ²1) is a potent inducer of chondrogenesis and stimulant of cartilage extracellular matrix (ECM) synthesis. Tissue inhibitor of metalloproteinasesβ3 (TIMPβ3) is located in ECM and is the major inhibitor of matrix metalloproteinases (MMPs) and aggrecanase,
β¦ LIBER β¦
Ciglitazone-induced p27 gene transcriptional activity is mediated through Sp1 and is negatively regulated by the MAPK signaling pathway
β Scribed by Fei Chen; Eugene Kim; Chia-Chi Wang; Lawrence E. Harrison
- Book ID
- 113527784
- Publisher
- Elsevier Science
- Year
- 2005
- Tongue
- English
- Weight
- 150 KB
- Volume
- 17
- Category
- Article
- ISSN
- 0898-6568
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
TGF-Ξ²-induced expression of tissue inhib
β
Hamid Yaqoob Qureshi; Judith Sylvester; Mohammed El Mabrouk; Muhammad Zafarullah
π
Article
π
2005
π
John Wiley and Sons
π
English
β 422 KB
MS34 HNE-INDUCED 5-LOX EXPRESSION IS TRA
β
Lee, S.-J.; Kim, C.-E.; Seo, K.-W.; Kim, C.-D.
π
Article
π
2010
π
Elsevier Science
π
English
β 58 KB
Activation of Stat1 and subsequent trans
β
Tadashi Nishiya; Takashi Uehara; Hiroki Edamatsu; Yoshito Kaziro; Hiroshi Itoh;
π
Article
π
1997
π
Elsevier Science
π
English
β 820 KB
Suppression of IL-8 gene expression by r
β
Yong Ju Na; Young Jin Jeon; Jae-Hong Suh; Jong Soon Kang; Kyu-Hwan Yang; Hwan-Mo
π
Article
π
2001
π
Elsevier Science
π
English
β 262 KB
Expression of the antiapoptotic MCL1 gen
β
Townsend, Karen J; Trusty, Jennifer L; Traupman, Matthew A; Eastman, Alan; Craig
π
Article
π
1998
π
Nature Publishing Group
π
English
β 488 KB
Vascular smooth muscle cell-specific reg
β
Mary M. Kavurma; Levon M. Khachigian
π
Article
π
2004
π
John Wiley and Sons
π
English
β 323 KB
π 1 views
## Abstract Smooth muscle cells (SMC) play a central role in common vascular pathologies such as atherosclerosis and restenosis. Understanding the molecular regulation of SMC proliferation at a transcriptional level may provide important clues for the targeted control of vascular hyperplasia. We re