D. melanogaster development was markedly retarded and its survival decreased by larvae treatment with compounds being strong inducers of the cytochrome P-450 2B in mammalsphenobarbital (PB\*), perfluorodecaline (PFD), transstilbene oxide (TSO), and triphenyldioxane (TPD). At the same time, the weak
โฆ LIBER โฆ
Chitin biosynthesis during pupal development of Drosophila melanogaster and the effect of thelethalcryptocephal mutation
โ Scribed by Sparrow, John C. ;Chadfield, Colin G.
- Publisher
- John Wiley and Sons
- Year
- 1982
- Tongue
- English
- Weight
- 632 KB
- Volume
- 3
- Category
- Article
- ISSN
- 0192-253X
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## Abstract In order to gain a better understanding of how a defect in pyrimidine biosynthesis leads to the specific set of morphological aberrations seen in __Drosophila__ bearing the mutant allele of __rudimentary__, we made the following measurements of enzyme activity in wild type flies: (1) du