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Chemotherapy for minimally differentiated acute myeloid leukemia (AML-M0)

โœ Scribed by N. Yokose; K. Ogata; T. Ito; K. Miyake; E. An; K. Inokuchi; T. Yamada; S. Gomi; Y. Tanabe; I. Ohki; T. Kuwabara; S. Hasegawa; T. Shinohara; K. Dan; T. Nomura

Publisher
Springer
Year
1993
Tongue
English
Weight
394 KB
Volume
66
Category
Article
ISSN
0939-5555

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## Abstract The __AML1__ gene encodes a transcription factor that, together with its heterodimeric partner CBFB, regulates a number of target genes that are essential for normal hemopoiesis. In acute myeloid leukemia (AML), __AML1__ is disrupted not only by chromosomal translocations but also by mu