𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Bone cell–matrix protein interactions

✍ Scribed by P. J. Marie

Publisher
Springer-Verlag
Year
2009
Tongue
English
Weight
120 KB
Volume
20
Category
Article
ISSN
0937-941X

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Endothelial cell–Matrix interactions
Endothelial cell–Matrix interactions
✍ Erika Iivanainen; Veli-Matti Kähäri; Jyrki Heino; Klaus Elenius 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 181 KB

## Abstract Dynamic interactions between endothelial cells and components of their surrounding extracellular matrix are necessary for the invasion, migration, and survival of endothelial cells during angiogenesis. These interactions are mediated by matrix receptors that initiate intracellular signa

Analyzing protein–protein interactions i
Analyzing protein–protein interactions in cell membranes
✍ Anja Nohe; Nils O. Petersen 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 287 KB 👁 1 views

## Abstract Interactions among membrane proteins regulate numerous cellular processes, including cell growth, cell differentiation and apoptosis. We need to understand which proteins interact, where they interact and to which extent they interact. This article describes a set of novel approaches to

Cell-matrix interactions during tumor in
Cell-matrix interactions during tumor invasion
✍ Jean R. Starkey 📂 Article 📅 1990 🏛 Springer 🌐 English ⚖ 982 KB

This manuscript reviews the molecular aspects of tumor cell invasion of extracellular matrix. The changes in cell:substrate and cell:cell receptors that characterize motile cells are discussed for their importance not only in mediating invasive cell behavior, but also as diagnostic markers for invas

Functional domains of necdin for protein
Functional domains of necdin for protein–protein interaction, nuclear matrix targeting, and cell growth suppression
✍ Hideo Taniura; Masakatsu Kobayashi; Kazuaki Yoshikawa 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 430 KB

## Abstract Necdin is a growth suppressor expressed predominantly in postmitotic neurons. The necdin gene is involved in the etiology of the genomic imprinting‐associated neurodevelopmental disorder Prader–Willi syndrome and belongs to the MAGE gene family. All the MAGE family proteins contain a la