✦ LIBER ✦

BH3-only proteins in cell death initiation, malignant disease and anticancer therapy

✍ Scribed by Labi, V; Erlacher, M; Kiessling, S; Villunger, A

Book ID: 110017239
Publisher: Nature Publishing Group
Year: 2006
Tongue: English
Weight: 219 KB
Volume: 13
Category: Article
ISSN: 1350-9047
DOI: 10.1038/sj.cdd.4401940

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

BH3-only proteins and their roles in pro

BH3-only proteins and their roles in programmed cell death

✍ Giam, M; Huang, D C S; Bouillet, P 📂 Article 📅 2008 🏛 Nature Publishing Group 🌐 English ⚖ 288 KB

BH3-only proteins in tumorigenesis and m

BH3-only proteins in tumorigenesis and malignant melanoma

✍ A. M. Karst; G. Li 📂 Article 📅 2006 🏛 Springer 🌐 English ⚖ 232 KB

BH3-only protein BIK induces caspase-ind

BH3-only protein BIK induces caspase-independent cell death with autophagic features in Bcl-2 null cells

✍ Rashmi, R; Pillai, S G; Vijayalingam, S; Ryerse, J; Chinnadurai, G 📂 Article 📅 2007 🏛 Nature Publishing Group 🌐 English ⚖ 363 KB

BIK, the founding member of the BH3-only

BIK, the founding member of the BH3-only family proteins: mechanisms of cell death and role in cancer and pathogenic processes

✍ Chinnadurai, G; Vijayalingam, S; Rashmi, R 📂 Article 📅 2008 🏛 Nature Publishing Group 🌐 English ⚖ 357 KB

In vivo contributions of BH3-only protei

In vivo contributions of BH3-only proteins to neuronal death following seizures, ischemia, and traumatic brain injury

✍ Engel, Tobias; Plesnila, Nikolaus; Prehn, Jochen H M; Henshall, David C 📂 Article 📅 2011 🏛 Nature Publishing Group 🌐 English ⚖ 655 KB

BH3-only proteins Bid and BimEL are diff

BH3-only proteins Bid and BimEL are differentially involved in neuronal dysfunction in mouse models of Huntington's disease

✍ Juan M. García-Martínez; Esther Pérez-Navarro; Xavier Xifró; Josep M. Canals; Mi 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 731 KB

## Abstract Apoptosis, a cell death mechanism regulated by Bcl‐2 family members, has been proposed as one of the mechanisms leading to neuronal loss in Huntington's disease (HD). Here we examined the regulation of Bcl‐2 family proteins in three different mouse models of HD with exon 1 mutant huntin