𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Beta-lapachone inhibits proliferation and induces apoptosis in retinoblastoma cell lines

✍ Scribed by Shah, H R; Conway, R M; Van Quill, K R; Madigan, M C; Howard, S A; Qi, J; Weinberg, V; O'Brien, J M

Book ID
110036845
Publisher
Nature Publishing Group
Year
2007
Tongue
English
Weight
131 KB
Volume
22
Category
Article
ISSN
0950-222X

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

GCP, a genistein-rich compound, inhibits
GCP, a genistein-rich compound, inhibits proliferation and induces apoptosis in lymphoma cell lines
✍ Jamie L. McCall; Rebekah A. Burich; Philip C. Mack πŸ“‚ Article πŸ“… 2010 πŸ› Elsevier Science 🌐 English βš– 1004 KB

Genistein combined polysaccharide (GCP), derived from soy bean extract, is comprised of deglycosylated isoflavones, such as genistein and daidzein. The goal of this work was to determine if GCP is effective in lymphoid cancers. In three human and four canine lymphoid cell lines, GCP inhibited prolif

Paclitaxel and beta-lapachone synergisti
Paclitaxel and beta-lapachone synergistically induce apoptosis in human retinoblastoma Y79 cells by downregulating the levels of phospho-Akt
✍ Antonella D'Anneo; Giuseppa Augello; Andrea Santulli; Michela Giuliano; Riccardo πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 454 KB

## Abstract Paclitaxel (PTX) and beta‐lapachone (LPC) are naturally occurring compounds that have shown a large spectrum of anticancer activity. In this article we show for the first time that PTX/LPC combination induces potent synergistic apoptotic effects in human retinoblastoma Y79 cells. Combin

Knockdown of TSP50 inhibits cell prolife
Knockdown of TSP50 inhibits cell proliferation and induces apoptosis in P19 cells
✍ Liang-Zhou; Yong-Li Bao; Yu Zhang; Yin Wu; Chun-Lei Yu; Yan-Xin Huang; Ying Sun; πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 365 KB

## Abstract Earlier studies identified testes‐specific protease 50 (__TSP50__), which encodes a threonine protease, and showed that it was abnormally reactivated in many breast cancer biopsies. Further, it was shown to be negatively regulated by the __p53__ gene. However, little is known about the