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Ataxia-telangiectasia-mutated dependent phosphorylation of Artemis in response to DNA damage

✍ Scribed by Ling Chen; Tomohiro Morio; Yoshiyuki Minegishi; Shin-Ichiro Nakada; Masayuki Nagasawa; Kenshi Komatsu; Luciana Chessa; Anna Villa; Daniele Lecis; Domenico Delia; Shuki Mizutani

Book ID
108583461
Publisher
John Wiley and Sons
Year
2005
Tongue
English
Weight
271 KB
Volume
96
Category
Article
ISSN
1347-9032

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## Abstract In response to certain types of DNA damage, ataxia telangiectasia and rad3‐related (ATR) phosphorylates checkpoint kinase 1 (CHEK1) resulting in cell cycle arrest and subsequent DNA repair. __ATR__ and __CHEK1__ contain mononucleotide microsatellite repeat regions, which are mutational