Genetic host factors may modify the course of the hepatitis C virus (HCV) infection. Very recently, a genome-wide scan that reported association of the IL28B locus with response to treatment in HCV infection was published. The aim of the current study was to investigate the relationship of this locu
β¦ LIBER β¦
Association between interleukin-28B-related genetic variants and liver histopathology differs between hepatitis C virus genotypes
β Scribed by Karolina Rembeck; Johan Westin; Magnus Lindh; Kristoffer Hellstrand; Gunnar Norkrans; Martin Laging; For the NORDynamIC study group
- Book ID
- 112097846
- Publisher
- John Wiley and Sons
- Year
- 2012
- Tongue
- English
- Weight
- 833 KB
- Volume
- 56
- Category
- Article
- ISSN
- 0270-9139
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Interleukin-28B genetic variants and hep
β
Marco Antonio Montes-Cano; JosΓ© RaΓΊl GarcΓa-Lozano; Cristina Abad-Molina; Manuel
π
Article
π
2010
π
John Wiley and Sons
π
English
β 107 KB
π 1 views
A Model Explaining the Correlations Betw
β
Magnus Lindh; Martin Lagging; Gunnar Norkrans; Kristoffer Hellstrand
π
Article
π
2010
π
Elsevier Science
π
English
β 328 KB
1182 THE LIVER LYMPHOCYTE INTRA-HEPATIC
β
Zarski, J.P.; ThΓ©lu, M.A.; Marche, H.; Fugier, E.; Marlu, A.; Sturm, N.; Leroy,
π
Article
π
2013
π
Elsevier Science
π
English
β 87 KB
Interleukin 28B polymorphisms are the on
β
P. J. Clark; A. J. Thompson; M. Zhu; D. M. Vock; Q. Zhu; D. Ge; K. Patel; S. A.
π
Article
π
2012
π
John Wiley and Sons
π
English
β 185 KB
Lack of association between occult hepat
β
KEI FUJIWARA; YASUHITO TANAKA; ETSURO ORITO; TOMOYOSHI OHNO; TAKANOBU KATO; FUMI
π
Article
π
2004
π
John Wiley and Sons
π
English
β 207 KB
Relationship between the interleukin-28b
β
Dennis Eurich; Sabine Boas-Knoop; Martin Ruehl; Maria Schulz; Esperanza D. Carri
π
Article
π
2011
π
John Wiley and Sons
π
English
β 293 KB
Up to 30% of liver transplants will develop graft cirrhosis within 5 years after liver transplantation (LT) due to recurrent HCV-infection forwarding accelerated graft damage. Genetic variants of cytokines involved in the immune response may contribute to the degree of graft inflammation, fibrosis p