𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Aspirin causes in vivo synthesis of type i collagen by atrophic articular cartilage

✍ Scribed by Renate E. Gay; Marshall J. Palmoski; Kenneth D. Brandt; Steffen Gay

Publisher
John Wiley and Sons
Year
1983
Tongue
English
Weight
556 KB
Volume
26
Category
Article
ISSN
0004-3591

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Mutations in fibrillar collagens (types
Mutations in fibrillar collagens (types I, II, III, and XI), fibril-associated collagen (type IX), and network-forming collagen (type X) cause a spectrum of diseases of bone, cartilage, and blood vessels
✍ Helena Kuivaniemi; Gerard Tromp; Darwin J. Prockop πŸ“‚ Article πŸ“… 1997 πŸ› John Wiley and Sons 🌐 English βš– 218 KB πŸ‘ 2 views

This review summarizes the data on 278 different mutations found to date in the genes for types I, II, III, IX, X, and XI collagens from 317 apparently unrelated patients. A majority (217 mutations; 78% of the total) of the mutations are single-base and either change the codon of a critical amino ac

Transfer of proΞ±2(I) cDNA into cells of
Transfer of proΞ±2(I) cDNA into cells of a murine model of human Osteogenesis Imperfecta restores synthesis of type I collagen comprised of Ξ±1(I) and Ξ±2(I) heterotrimers in vitro and in vivo
✍ Christopher Niyibizi; Patrick Smith; Zhibao Mi; Charlotte L. Phillips; Paul Robb πŸ“‚ Article πŸ“… 2001 πŸ› John Wiley and Sons 🌐 English βš– 197 KB

## Abstract The __oim__ mouse is a model of human Osteogenesis Imperfecta (OI) that has deficient synthesis of proΞ±2(I) chains. Cells isolated from __oim__ mice synthesize Ξ±1(I) collagen homotrimers that accumulate in tissues. To explore the feasibility of gene therapy for OI, a murine proΞ±2(I) cDN