๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Amyloid transfer from a syngeneic graft of amyloid spleen in intact and amyloid mice

โœ Scribed by V. V. Sura; I. N. Osipova; A. P. Chebyshev; V. V. Serov; A. Yu. Gritsman

Publisher
Springer US
Year
1978
Tongue
English
Weight
678 KB
Volume
85
Category
Article
ISSN
0007-4888

No coin nor oath required. For personal study only.

๐Ÿ“œ SIMILAR VOLUMES

Modeling behavioral and neuronal symptom
Modeling behavioral and neuronal symptoms of Alzheimer's disease in mice: A role for intraneuronal amyloid
โœ L. Gimรฉnez-Llort; G. Blรกzquez; T. Caรฑete; B. Johansson; S. Oddo; A. Tobeรฑa; F.M. ๐Ÿ“‚ Article ๐Ÿ“… 2007 ๐Ÿ› Elsevier Science ๐ŸŒ English โš– 633 KB

The amyloid Abeta-peptide (Abeta) is suspected to play a critical role in the cascade leading to AD as the pathogen that causes neuronal and synaptic dysfunction and, eventually, cell death. Therefore, it has been the subject of a huge number of clinical and basic research studies on this disease. A

In vivo visualization of Alzheimer's amy
In vivo visualization of Alzheimer's amyloid plaques by magnetic resonance imaging in transgenic mice without a contrast agent
โœ Clifford R. Jack Jr.; Michael Garwood; Thomas M. Wengenack; Bret Borowski; Geoff ๐Ÿ“‚ Article ๐Ÿ“… 2004 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 800 KB

## Abstract One of the cardinal pathologic features of Alzheimer's disease (AD) is the formation of senile, or amyloid, plaques. Transgenic mice have been developed that express one or more of the genes responsible for familial AD in humans. Doubly transgenic mice develop โ€œhumanโ€likeโ€ plaques, prov