𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Activation of ERK5 in angiotensin II-induced hypertrophy of human aortic smooth muscle cells

✍ Scribed by Zhuo Zhao; Jing Geng; Zhiming Ge; Wei Wang; Yun Zhang; Weiqiang Kang

Publisher
Springer
Year
2008
Tongue
English
Weight
377 KB
Volume
322
Category
Article
ISSN
0300-8177

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Determinants of angiotensin II-induced h
Determinants of angiotensin II-induced hypertrophy versus hyperplasia in vascular smooth muscle
✍ Gary K. Owens πŸ“‚ Article πŸ“… 1993 πŸ› John Wiley and Sons 🌐 English βš– 536 KB

There is clear evidence implicating a role for angiotensin II in control of hypertrophic growth of vascular smooth muscle cells in vivo. However, the specific mechanisms whereby angiotensin II influences smooth muscle cell growth have not been defined. The focus of this mini-review is to consider h

Cytoskeletal remodeling in vascular smoo
Cytoskeletal remodeling in vascular smooth muscle cells in response to angiotensin II-induced activation of the SHP-2 tyrosine phosphatase
✍ Karl Fernstrom; Paul Farmer; M. Showkat Ali πŸ“‚ Article πŸ“… 2005 πŸ› John Wiley and Sons 🌐 English βš– 541 KB

## Abstract Angiotensin II is an octapeptide that regulates diverse cellular responses including the actin cytoskeletal organization. In this study, stable cell lines overexpressing wild‐type or catalytically inactive SHP‐2 were employed to elucidate the signaling pathway utilized by the SHP‐2 tyro

Insulin enhances angiotensin II induced
Insulin enhances angiotensin II induced DNA synthesis in vascular smooth muscle cells of the rat
✍ Ko, Y. ;Sachinidis, A. ;Wieczorek, A.J. ;Appenheimer, M. ;DοΏ½sing, R. ;Vetter, H. πŸ“‚ Article πŸ“… 1993 πŸ› Springer-Verlag 🌐 English βš– 401 KB

Hypertension has a high prevalence among subjects with decreased insulin sensitivity and/or hyperinsulinemia. Furthermore, angiotensin II plays a pivotal role in the regulation of vascular tone and is known to induce hypertrophy and/or hyperplasia in vascular smooth muscle cells. In the present stud