The effects of bath-applied sodium nitroprusside (SNP), a nitric oxide (NO) donor, on an acetylcholine (ACh)-induced K+ current recorded from identified neurons (R9 and R10) of Aplysia kurodui were investigated with conventional voltage-clamp and pressure ejection techniques. Bath-applied SNP (25-50
255 Nitric oxide inhibits the ach-induced K+ cueeent in identified Aplysia neurons
โ Scribed by Masashi Sawada; Mitsuyuki Ichinose
- Book ID
- 119130181
- Publisher
- Elsevier Science
- Year
- 1996
- Tongue
- English
- Weight
- 100 KB
- Volume
- 25
- Category
- Article
- ISSN
- 0168-0102
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๐ SIMILAR VOLUMES
Nitric oxide (NO) is produced by the enzyme nitric oxide synthase (NOS) and has been implicated in inter- and intracellular communication in the nervous system. The present study was undertaken to assess the effects of sodium nitroprusside (SNP) and hydroxylamine (HOA), NO donors, on a dopamine (DA)
The effects of extracellularly applied recombinant human interleukin-2 (rhIL-2) on the y-aminobutyric acid (GABA)-induced C1-current recorded from identified neurons (R9 and R12) of Aplysia kurodui were investigated with conventional voltage-clamp and pressure ejection techniques. Bath-applied rhIL-
Nitric oxide (NO) acts as a neurotransmitter and neuromodulator in the nervous system of many vertebrates and invertebrates. The effects of extracellularly applied sodium nitroprusside (SNP) and diethylamine NO (C(2)H(5))(2)N[N(O)NO]-Na(+) (DEA/NO), NO donors, on a glutamate (Glu)-induced K(+) curre