✦ LIBER ✦

2,3-Butanedione 2-Monoxime (BDM) Induces Calcium Release from Canine Cardiac Sarcoplasmic Reticulum

✍ Scribed by Ronald M. Phillips; Ruth A. Altschuld

Book ID: 115579427
Publisher: Elsevier Science
Year: 1996
Tongue: English
Weight: 79 KB
Volume: 229
Category: Article
ISSN: 0006-291X
DOI: 10.1006/bbrc.1996.1772

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

2,3-Butanedione monoxime (BDM) decreases

2,3-Butanedione monoxime (BDM) decreases sarcoplasmic reticulum Ca content by stimulating Ca release in isolated rat ventricular myocytes

✍ W. Adams; A. W. Trafford; D. A. Eisner 📂 Article 📅 1998 🏛 Springer 🌐 English ⚖ 88 KB

Reduction in the Ca2+-induced Ca2+ relea

Reduction in the Ca2+-induced Ca2+ release from canine cardiac sarcoplasmic reticulum following endotoxin administration

✍ Maw-Shung Liu; Li-Ling Wu 📂 Article 📅 1991 🏛 Elsevier Science 🌐 English ⚖ 435 KB

9-Methyl-7-bromoeudistomin D induces Ca2

9-Methyl-7-bromoeudistomin D induces Ca2+ release from cardiac sarcoplasmic reticulum

✍ Asami Seino-Umeda; Yan-Il Fang; Masami Ishibashi; Jun'ichi Kobayashi; Yasushi Oh 📂 Article 📅 1998 🏛 Elsevier Science 🌐 English ⚖ 111 KB

Effects of caffeine and 2,3-butanedione

Effects of caffeine and 2,3-butanedione monoxime (BDM) on contractile properties of skinned cardiac fibres from normal and diabetic rats

✍ N. Khandoudi; J.M. Chesnais; D. Feuvray 📂 Article 📅 1990 🏛 Elsevier Science 🌐 English ⚖ 97 KB

Contractile activation and the effects o

Contractile activation and the effects of 2,3-butanedione monoxime (BDM) in skinned cardiac preparations from normal and dystrophic mice (129/ReJ)

✍ J. M. West; D. G. Stephenson 📂 Article 📅 1989 🏛 Springer 🌐 English ⚖ 870 KB

Inositol trisphosphate enhances Ca2+osci

Inositol trisphosphate enhances Ca2+oscillations but not Ca2+-induced Ca2+release from cardiac sarcoplasmic reticulum

✍ Yu Zhu; Thomas M. Nosek 📂 Article 📅 1991 🏛 Springer 🌐 English ⚖ 686 KB

The role of inositol 1,4,5-trisphosphate [Ins(1,4,5)P3] in excitation-contraction coupling in cardiac muscle is still unclear, although many laboratories are beginning to assume a critical role for this putative second messenger. Earlier studies from this laboratory [Nosek et al. (1986) Am J Physiol