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1,25(OH)2D3Induces a Mineralization Defect and Loss of Bone Mineral Density in Genetic Hypercalciuric Stone-Forming Rats

✍ Scribed by Ng, Adeline H.; Frick, Kevin K.; Krieger, Nancy S.; Asplin, John R.; Cohen-McFarlane, Madison; Culbertson, Christopher D.; Kyker-Snowman, Kelly; Grynpas, Marc D.; Bushinsky, David A.

Book ID
121561320
Publisher
Springer
Year
2014
Tongue
English
Weight
974 KB
Volume
94
Category
Article
ISSN
1432-0827

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## Abstract We have bred a strain of rats to maximize urine (u) calcium (Ca) excretion and model hypercalciuric nephrolithiasis. These genetic hypercalciuric stone‐forming (GHS) rats excrete more uCa than control Sprague‐Dawley rats, uniformly form kidney stones, and similar to patients, demonstrat