ΔF508 CFTR Localizes in the Endoplasmic Reticulum–Golgi Intermediate Compartment in Cystic Fibrosis Cells
✍ Scribed by Anne Gilbert; Michel Jadot; Ekaterina Leontieva; Simone Wattiaux-De Coninck; Robert Wattiaux
- Book ID
- 115602995
- Publisher
- Elsevier Science
- Year
- 1998
- Tongue
- English
- Weight
- 409 KB
- Volume
- 242
- Category
- Article
- ISSN
- 0014-4827
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
The recently identified Alzheimer's disease-associated presenilin 1 and 2 (PS1 and PS2) genes encode two homologous multi membrane-spanning proteins. Rabbit antibodies to the N-terminal domain of PS1 detected PS1 in human neuroblastoma SH-SY5Y wild type and PS1 transfectants (SY5Y-PS1) as well as in
Cystic fibrosis (CF) is the most common autosomal recessive disease in the Caucasian population. The disease can be caused by one of the more than 900 different mutations in the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) gene. However, the deletion of the phe508-codon is the most pre
The morphology of the mouse vas deferens still undergoes major changes from birth to 40 days of age, such as differentiation of the mesenchymal cells into fibroblasts and muscle cells, differentiation of the epithelium into basal and columnar epithelial cells, development of stereocilia, and the app