✦ LIBER ✦

Ventilator-induced lung injury leads to loss of alveolar and systemic compartmentalization of tumor necrosis factor-α

✍ Scribed by Jack J. Haitsma; Stefan Uhlig; Rolf Göggel; Serge J. Verbrugge; Ulrike Lachmann; Burkhard Lachmann

Book ID: 105837725
Publisher: Springer
Year: 2000
Tongue: English
Weight: 88 KB
Volume: 26
Category: Article
ISSN: 1432-1238
DOI: 10.1007/s001340000648

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Resistance to silica-induced lung fibros

Resistance to silica-induced lung fibrosis in senescent rats: role of alveolar macrophages and tumor necrosis factor-α (TNF)

✍ E. Corsini; A. Giani; S. Peano; M. Marinovich; C.L. Galli 📂 Article 📅 2004 🏛 Elsevier Science 🌐 English ⚖ 48 KB

Influence of hyperbaric oxygen on tumor

Influence of hyperbaric oxygen on tumor necrosis factor-α and nitric oxide production in endotoxin-induced acute lung injury in rats

✍ Shi-Jye Chu; Min-Hui Li; Ching-Wang Hsu; Shih-Hung Tsai; Shih-Hua Lin; Kun-Lun H 📂 Article 📅 2007 🏛 Elsevier Science 🌐 English ⚖ 372 KB

Bone morphogenetic protein-7 antagonizes

Bone morphogenetic protein-7 antagonizes tumor necrosis factor-α–induced activation of nuclear factor κB and up-regulation of the ADAMTS, leading to decreased degradation of disc matrix macromolecules aggrecan and collagen II

✍ Wang, Zili; Hutton, William C.; Yoon, S. Tim 📂 Article 📅 2014 🏛 Elsevier Science 🌐 English ⚖ 853 KB

Interleukin-1β (IL-1β)-induced modulatio

Interleukin-1β (IL-1β)-induced modulation of the hypothalamic IL-1β system, tumor necrosis factor-α, and transforming growth factor-β1 mRNAs in obese (fa/fa) and lean (Fa/Fa) Zucker rats: Implications to IL-1β feedback systems and cytokine–cytokine interactions

✍ Carlos R. Plata-Salamán; Sergey E. Ilyin 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 111 KB 👁 1 views

## Interleukin -1␤ (IL-1␤) induces anorexia, fever, sleep changes, and neuroendocrine alterations when administered into the brain. Here, we investigated the regulation of the IL-1␤ system (ligand, receptors, receptor accessory protein, and receptor antagonist), tumor necrosis factor-␣ (TNF-␣), tr