## Abstract Nitric oxide (NO) and its metabolites are implicated in carcinogenesis and metastasis. Both stimulatory and inhibitory effects of NO have been reported in relation to breast cancer and its role in the development of malignancies and metastasis remains uncertain. We have used the polyoma
Vasopressin release during endotoxaemic shock in mice lacking inducible nitric oxide synthase
✍ Scribed by Evelin C. Carnio; Angelita M. Stabile; Marcelo E. Batalhão; João Santana Silva; José Antunes-Rodrigues; Luiz G. S. Branco; Sheldon Magder
- Publisher
- Springer
- Year
- 2005
- Tongue
- English
- Weight
- 229 KB
- Volume
- 450
- Category
- Article
- ISSN
- 0031-6768
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Lack of inducible nitric oxide synthase activity in T cell clones and T lymphocytes from naive and Leishmania major-infected mice Nitric oxide (NO) generated by the inducible isoform of nitric oxide synthase (iNOS) is implicated in a number of immunological processes including killing of intrac
The role of nitric oxide (NO) in liver injury and fibrosis is unclear. The purpose of this study was to determine whether inducible NO synthase deficiency (iNOS(-/-)) affects liver injury and fibrosis produced in mice by chronic carbon tetrachloride (CCl(4)) administration. Wild-type (WT) or iNOS(-/