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Tumor refractoriness to anti-VEGF treatment is mediated by CD11b+Gr1+ myeloid cells

✍ Scribed by Shojaei, Farbod; Wu, Xiumin; Malik, Ajay K; Zhong, Cuiling; Baldwin, Megan E; Schanz, Stefanie; Fuh, Germaine; Gerber, Hans-Peter; Ferrara, Napoleone


Book ID
109910964
Publisher
Nature Publishing Group
Year
2007
Tongue
English
Weight
383 KB
Volume
25
Category
Article
ISSN
1087-0156

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Epstein-Barr virus (EBV) can induce uncontrolled B lymphocyte proliferation leading to fatal lymphoma in immunocompromised patients. The sensitivity to apoptosis of B lymphoid cell lines (LCL) derived from EBV-induced lymphoproliferative disorders was investigated. In vitro and in vivo, these B LCL