𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Tumor Necrosis Factor-α, Interleukin-1β and Nitric Oxide: Induction of Liver Megamitochondria in Prehepatic Portal Hypertensive Rats

✍ Scribed by Isabel Prieto; Fulgencio Jiménez; Maria-Angeles Aller; Maria-Paz Nava; Elena Vara; Cruz Garcia; Jaime Arias

Publisher
Springer
Year
2005
Tongue
English
Weight
146 KB
Volume
29
Category
Article
ISSN
0364-2313

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Tumor necrosis factor α regulates nitric
Tumor necrosis factor α regulates nitric oxide synthase expression in portal hypertensive gastric mucosa of rats
✍ Masayuki Ohta; Andrzej S. Tarnawski; Rabiha Itani; Rama Pai; Morimasa Tomikawa; 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 185 KB 👁 1 views

Anti-tumor necrosis factor ␣ (TNF-␣) treatment decreases nitric oxide (NO) synthesis and ameliorates the hyperdynamic circulation in portal hypertensive rats. We have recently demonstrated that nitric oxide synthase isoform 3 (NOS3) is overexpressed in portal hypertensive gastric mucosa and that res

Thalidomide inhibits tumor necrosis fact
Thalidomide inhibits tumor necrosis factor α, decreases nitric oxide synthesis, and ameliorates the hyperdynamic circulatory syndrome in portal-hypertensive rats
✍ J C Lopez-Talavera; G Cadelina; J Olchowski; W Merrill; R J Groszmann 📂 Article 📅 1996 🏛 John Wiley and Sons 🌐 English ⚖ 281 KB

Portal hypertension accompanied by anatomic or served in portal hypertension with liver failure or extenfunctional portal-systemic shunting very often is assosive portal-systemic shunting. Tumor necrosis factor a ciated with a hyperdynamic circulatory state. The so-(TNF) causes marked hypotension in

Tumor necrosis factor α and interleukin-
Tumor necrosis factor α and interleukin-1β modulate calcium and nitric oxide signaling in mechanically stimulated osteocytes
✍ A. D. Bakker; V. C. da Silva; R. Krishnan; R. G. Bacabac; M. E. Blaauboer; Y.-C. 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 437 KB

## Abstract ## Objective Inflammatory diseases often coincide with reduced bone mass. Mechanoresponsive osteocytes regulate bone mass by maintaining the balance between bone formation and resorption. Despite its biologic significance, the effect of inflammation on osteocyte mechanoresponsiveness i