This study examines whether tumor necrosis factor and endotoxin are involved in the pathogenesis of primary nonfunction of graft and pulmonary complication after orthotopic liver transplantation. Livers from Lewis rats were stored for either 1 or 4 hr in ice-cold Em-Collins solution (1-hr storage an
Tumor necrosis factor, the acute phase response and the pathogenesis of alcoholic liver disease
โ Scribed by Dwain L. Thiele
- Publisher
- John Wiley and Sons
- Year
- 1989
- Tongue
- English
- Weight
- 377 KB
- Volume
- 9
- Category
- Article
- ISSN
- 0270-9139
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๐ SIMILAR VOLUMES
Acute graft-versus-host disease following orthotopic liver transplantation is a rare but feared complication arising in 1% to 2% of cases with a dismal prognosis. It most often presents as fever, rash, and diarrhea with or without pancytopenia. Patients die from complications of marrow failure such
We investigated the potential of dietary saturated fatty acids to decrease endotoxemia and suppress expression of cyclooxygenase 2 (Cox-2) and tumor necrosis factor alpha (TNF-alpha) in established alcohol-induced liver injury. Six groups (five rats/group) of male Wistar rats were studied. Rats in g
Background: Hepatitis C-related liver failure is the leading indication for liver transplantation worldwide. Although histologic recurrence is identified in the majority of patients the spectrum of allograft injury is wide. To date, most studies have focused on the contribution of immunosuppression