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Tuberous sclerosis complex 2 loss-of-function mutation regulates reactive oxygen species production through Rac1 activation

✍ Scribed by Tsukasa Suzuki; Swadesh K. Das; Hirohumi Inoue; Machiko Kazami; Okio Hino; Toshiyuki Kobayashi; Raymond S. Yeung; Ken-Ichi Kobayashi; Tadahiro Tadokoro; Yuji Yamamoto

Book ID
116296321
Publisher
Elsevier Science
Year
2008
Tongue
English
Weight
601 KB
Volume
368
Category
Article
ISSN
0006-291X

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## Abstract We investigated the effects of overexpression of the tuberous sclerosis‐1 and ‐2 (__TSC1/2__) gene products (hamartin and tuberin, respectively) in the human kidney epithelial cell line 293 with an inducible expression system. As we had observed previously in fibroblasts, 293 cells over