A growing body of experimental evidence demonstrates that the serpin antichymotrypsin plays a regulatory role in Alzheimer plaque physiology by interacting with the 42 residue p-amyloid protein, and we have used molecular modeling and energy minimization techniques to study this interaction. Based o
✦ LIBER ✦
Trypsin interaction with the senile plaques of Alzheimer disease is mediated by β-protein precursor
✍ Scribed by Mark A. Smith; Colleen E. Dunbar; Edward J. Miller; George Perry
- Book ID
- 105619912
- Publisher
- Springer-Verlag
- Year
- 1996
- Tongue
- English
- Weight
- 883 KB
- Volume
- 27
- Category
- Article
- ISSN
- 1044-7393
No coin nor oath required. For personal study only.
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