𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Transforming growth factor α activates Ha-Ras in human pancreatic cancer cells with Ki-ras mutations

✍ Scribed by Thomas Seufferlein*; Johan Van Lint‡; Susanne Liptay§; Guido Adler*; Roland M. Schmid*


Book ID
118522101
Publisher
Elsevier Science
Year
1999
Tongue
English
Weight
230 KB
Volume
116
Category
Article
ISSN
0016-5085

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Suppression of Ki-ras p21 levels leading
✍ Kazunori Aoki; Teruhiko Yoshida; Nobuyuki Matsumoto; Hisamitsu Ide; Takashi Sugi 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 168 KB 👁 1 views

Ki-ras point mutation characteristically occurs frequently in human pancreatic cancer. To clarify the effect of antisense Ki-ras RNA on various pancreatic cancer cell lines, a plasmid expressing an antisense Ki-ras gene fragment (AS-K-ras-LNSX) was transduced into seven human pancreatic cancer cell

Regulation of surface-differentiation mo
✍ F. Basolo; C. Serra; F. Ciardiello; L. Fiore; J. Russo; D. Campani; A. Dolei; F. 📂 Article 📅 1992 🏛 John Wiley and Sons 🌐 French ⚖ 770 KB

## Abstract Spontaneously immortalized human mammary epithelial cells MCF‐IOA were transfected with an activated c‐Ha‐ras oncogene. Transfected cells (MCF‐IOT) acquire a malignant pheno‐type, as already reported. Studies of l‐2′‐deoxyuridine incorporation in cultures given graded doses of hydrocort