𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Transcriptional regulation of livin by β-catenin/TCF signaling in human lung cancer cell lines

✍ Scribed by Dong Yuan; Liqun Liu; Dayong Gu


Publisher
Springer
Year
2007
Tongue
English
Weight
373 KB
Volume
306
Category
Article
ISSN
0300-8177

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Transactivation of MCP-1/CCL2 by β-caten
✍ Melanie Mestdagt; Myriam Polette; Giovanna Buttice; Agnes Noël; Atsuhisa Ueda; J 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 French ⚖ 296 KB

## Abstract The loss of E‐cadherin expression and the translocation of β‐catenin to the nucleus are frequently associated with the metastatic conversion of epithelial cells. In the nucleus, β‐catenin binds to the TCF/LEF‐1 (T‐cell factor/ lymphoid enhancer factor) transcription factor family result

β-Catenin transcriptional activity is in
✍ B. Kiely; R.T. O'Donovan; S.L. McKenna; G.C. O'Sullivan 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 French ⚖ 339 KB

## Abstract Aberrant expression/localisation of β‐catenin has been implicated in the progression of oesophageal cancer. As a member of the Wnt‐signalling pathway, activated β‐catenin translocates into the nucleus and drives gene transcription. Insulin‐like growth factors (IGFs) have been implicated

Regulation of RARβ expression by RAR- an
✍ Yin Li; Marcia I. Dawson; Anissa Agadir; Mi-Ock Lee; Ling Jong; Peter D. Hobbs; 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 French ⚖ 192 KB 👁 1 views

Retinoids regulate the growth and differentiation of human tracheobronchial epithelial cells. In this study, we investigated the effects of all-trans-retinoic acid (trans-RA) and receptor class-selective retinoids on the growth and apoptosis of human lung cancer cell lines. Trans-RA significantly in

Rap1 reverses transcriptional repression
✍ Kimberly D. Fralix; Shujie Zhao; Kolaparthi Venkatasubbarao; James W. Freeman 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 352 KB

## Abstract The TGF‐β signaling pathway has potent anti‐mitogenic effects in epithelial cells and loss of negative growth regulation is often associated with increased tumorigenicity. The human pancreatic ductal adenocarcinoma cell line, UK Pan‐1, which expresses __DPC4__, is not highly responsive