𝔖 Bobbio Scriptorium
✦   LIBER   ✦

TRANSCRIPTION OF Fcγ RECEPTORS IN DIFFERENT RAT LIVER CELLS

✍ Scribed by Torunn Løvdal; Trond Berg


Publisher
Elsevier Science
Year
2001
Tongue
English
Weight
239 KB
Volume
25
Category
Article
ISSN
1065-6995

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Receptor-mediated endocytosis of immune
✍ Seyed Ali Mousavi; Marita Sporstøl; Cathrine Fladeby; Rune Kjeken; Nicolas Baroi 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 794 KB

Liver sinusoidal endothelial cells (LSECs) display a number of receptors for efficient uptake of potentially injurious molecules. The receptors for the Fc portion of immunoglobulin G (IgG) antibodies (Fc␥Rs) regulate a number of physiological and pathophysiological events. We used reverse transcript

REGULATION OF RETINOID X RECEPTOR-γ GENE
✍ PANTELIS GEORGIADES; PAUL M. BRICKELL 📂 Article 📅 1998 🏛 Elsevier Science 🌐 English ⚖ 237 KB

Retinoid X receptor-(RXR ) is a transcription factor that mediates retinoid signalling and is expressed in rat heart during adult life. However, its expression in embryonic and neonatal heart has not been investigated and it is not known whether ventricular cardiomyocytes express RXR or whether all-

Different amplifying mechanisms of inter
✍ Lilyanne C. Grevers; Peter L. E. M. van Lent; Marije I. Koenders; Birgitte Walgr 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 341 KB 👁 1 views

## Abstract ## Objective Previously, we reported that interferon‐γ (IFNγ) aggravates cartilage destruction in immune complex (IC)–mediated arthritis via up‐regulation of activating Fcγ receptors (FcγR). Recently, we found that interleukin‐17 (IL‐17) also aggravates cartilage destruction in arthrit

TCDD-dependent downregulation of γ-caten
✍ Cornelia Dietrich; Dagmar Faust; Matthias Moskwa; Anja Kunz; Karl-Walter Bock; F 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 French ⚖ 306 KB

## Abstract TCDD (2,3,7,8‐tetrachlorodibenzo‐__p__‐dioxin) is the most potent tumor promoter ever tested in rodents. Although it is known that most of the effects of TCDD are mediated by binding to the aryl hydrocarbon receptor (AHR), the mechanisms leading to tumor promotion still remain to be elu