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Toxicity of Familial ALS-Linked SOD1 Mutants from Selective Recruitment to Spinal Mitochondria

✍ Scribed by Jian Liu; Concepción Lillo; P.Andreas Jonsson; Christine Vande Velde; Christopher M Ward; Timothy M Miller; Jamuna R Subramaniam; Jeffery D Rothstein; Stefan Marklund; Peter M Andersen; Thomas Brännström; Ole Gredal; Philip C Wong; David S Williams; Don W Cleveland

Book ID: 116781978
Publisher: Elsevier Science
Year: 2004
Tongue: English
Weight: 709 KB
Volume: 43
Category: Article
ISSN: 0896-6273
DOI: 10.1016/j.neuron.2004.06.016

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## Abstract Amyotrophic lateral sclerosis (ALS) is the most common fatal motor neuron disease, affecting mostly middle‐aged people. There are no curative therapies for ALS. Several lines of evidence have supported the notion that the proapoptotic property of familial ALS (FALS)‐linked mutant Cu/Zn‐