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The α-like scorpion toxin BmK I enhances membrane excitability via persistent sodium current by preventing slow inactivation and deactivation of rNav1.2a expressed in Xenopus Oocytes

✍ Scribed by Mang-Mang Zhu; Miao Tan; Hui-Wen Cheng; Yong-Hua Ji


Book ID
116919964
Publisher
Elsevier Science
Year
2009
Tongue
English
Weight
794 KB
Volume
23
Category
Article
ISSN
0887-2333

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