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The PTPN22 R263Q polymorphism is a risk factor for rheumatoid arthritis in Caucasian case–control samples

✍ Scribed by Luis Rodríguez-Rodríguez; Wan Rohani Wan Taib; Ruth Topless; Sophia Steer; María F. González-Escribano; Alejandro Balsa; Dora Pascual-Salcedo; Miguel A. González-Gay; Enrique Raya; Benjamín Fernandez-Gutierrez; Isidoro González-Álvaro; Nunzio Bottini; Torsten Witte; Marte K. Viken; Marieke J. H. Coenen; Piet L. C. M. van Riel; Barbara Franke; Martin den Heijer; Timothy R. D. J. Radstake; Paul Wordsworth; Benedicte A. Lie; Tony R. Merriman; Javier Martín

Publisher
John Wiley and Sons
Year
2011
Tongue
English
Weight
97 KB
Volume
63
Category
Article
ISSN
0004-3591

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The −169C/T polymorphism in FCRL3 is not
The −169C/T polymorphism in FCRL3 is not associated with susceptibility to rheumatoid arthritis or systemic lupus erythematosus in a case–control study of Koreans
✍ Chan-Bum Choi; Changsoo Paul Kang; Sang-Seokg Seong; Sang-Cheol Bae; Changwon Ka 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 57 KB 👁 2 views

## Abstract ## Objective In Japanese individuals, the −169C/T single‐nucleotide polymorphism (SNP) in __FCRL3__ has been reported to be associated with rheumatoid arthritis (RA), systemic lupus erythematosus (SLE), and autoimmune thyroid diseases. The objective of this study was to test the associ