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The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3α activity in diabetic myotubes

✍ Scribed by Michael Gaster; Klaus Brusgaard; Aase Handberg; Kurt Højlund; Jorgen F.P Wojtaszewski; Henning Beck-Nielsen

Book ID
116288613
Publisher
Elsevier Science
Year
2004
Tongue
English
Weight
284 KB
Volume
319
Category
Article
ISSN
0006-291X

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## Abstract We have evaluated the effect of peripheral insulin deficiency on brain insulin pathway activity in a mouse model of type 1 diabetes, the parallels with Alzheimer's disease (AD), and the effect of treatment with insulin. Nine weeks of insulin‐deficient diabetes significantly impaired the