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The presence of beta-amyloid 1-42 nicotinic acetylcholine receptors in an in vitro presynaptic model causes dysregulated calcium signaling, mitochondrial dysfunction and altered ERK signaling on sustained exposure to beta-amyloid

✍ Scribed by Arora, Komal; Alfulaij, Naghum; Nichols, Robert


Book ID
122045597
Publisher
Elsevier Science
Year
2012
Tongue
English
Weight
61 KB
Volume
8
Category
Article
ISSN
1552-5260

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