✦ LIBER ✦

The leukemia-associated transcription repressor AML1/MDS1/EVI1 requires CtBP to induce abnormal growth and differentiation of murine hematopoietic cells

✍ Scribed by Senyuk, Vitalyi; Chakraborty, Soumen; Mikhail, Fady M; Zhao, Rui; Chi, Yiqing; Nucifora, Giuseppina

Book ID: 110066630
Publisher: Nature Publishing Group
Year: 2002
Tongue: English
Weight: 389 KB
Volume: 21
Category: Article
ISSN: 0950-9232
DOI: 10.1038/sj.onc.1205436

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

MDS1/EVI1 enhances TGF-β1 signaling and

MDS1/EVI1 enhances TGF-β1 signaling and strengthens its growth-inhibitory effect, but the leukemia-associated fusion protein AML1/MDS1/EVI1, product of the t(3;21), abrogates growth-inhibition in response to TGF-β1

✍ Sood, R; Talwar-Trikha, A; Chakrabarti, SR; Nucifora, G 📂 Article 📅 1999 🏛 Nature Publishing Group 🌐 English ⚖ 216 KB

Resistance to 1,25D-induced differentiat

Resistance to 1,25D-induced differentiation in human acute myeloid leukemia HL60-40AF cells is associated with reduced transcriptional activity and nuclear localization of the vitamin D receptor

✍ Edward Garay; Robert Donnelly; Xuening Wang; George P. Studzinski 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 438 KB

## Abstract The anti‐neoplastic effects of 1,25‐dihydroxyvitamin D~3~ (1,25D) are well documented in numerous tumor cell systems and animal models of cancer. However, despite this pre‐clinical success, the clinical use of 1,25D is currently impeded by the dose‐limiting hypercalcemia, and the risk o