✦ LIBER ✦

The integrin signalling adaptor p130CAS is also a key player in prostate cancer

✍ Scribed by Fromont, Gaelle; Cussenot, Olivier

Book ID: 109948650
Publisher: Nature Publishing Group
Year: 2011
Tongue: English
Weight: 97 KB
Volume: 11
Category: Article
ISSN: 1474-1776
DOI: 10.1038/nrc2967-c1

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

THE Wnt SIGNALLING PATHWAY IS A POTENTIA

THE Wnt SIGNALLING PATHWAY IS A POTENTIAL THERAPEUTIC TARGET IN PROSTATE CANCER

✍ GEORGE W. YARDY; SIMON F. BREWSTER 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 67 KB

THE SEF LONG ISOFORM A IS A KEY INHIBITO

THE SEF LONG ISOFORM A IS A KEY INHIBITOR OF FGF INDUCED TUMOURIGENIC BEHAVIOUR THROUGH INHIBITION OF MULTIPLE SIGNALLING PATHWAYS IN PROSTATE CANCER

✍ S. Darby; C. Robson; H. Leung; V.J. Gnanapragasam 📂 Article 📅 2008 🏛 Elsevier Science 🌐 English ⚖ 70 KB

625 THE NEGATIVE SIGNALLING REGULATOR SP

625 THE NEGATIVE SIGNALLING REGULATOR SPRED 1 IS UP-REGULATED IN PROSTATE CANCER AND MODULATES THE EFFECTIVENESS OF TARGETED INHIBITION OF MAPK SIGNALLING

✍ J.A. Sewell; A. Joseph; T. Murphy; S. Hori; C.N. Robson; V.J. Gnanapragasam 📂 Article 📅 2011 🏛 Elsevier Science 🌐 English ⚖ 177 KB

45 POSTER Focal adhesion kinase is a key

45 POSTER Focal adhesion kinase is a key signalling intermediate in interleukin-8 promoted chemotaxis and adhesion of prostate cancer cells to bone marrow endothelium

✍ S. McFarlane; A. Seaton; A. Chacko; P.G. Johnston; R. Montironi; D.J. Waugh 📂 Article 📅 2006 🏛 Elsevier Science 🌐 English ⚖ 60 KB

EGF signalling in prostate cancer cell l

EGF signalling in prostate cancer cell lines is inhibited by a high expression level of the endocytosis protein REPS2

✍ Josien K. Oosterhoff; Liesbeth C. Kühne; J. Anton Grootegoed; Leen J. Blok 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 French ⚖ 286 KB 👁 2 views

In advanced prostate cancer, cellular changes occur leading to a transition from androgen-dependent to androgen-independent growth. During this transition, proliferation of androgendependent prostate cancer cells becomes more and more dependent on growth factors, like the epidermal growth factor (EG