The increased mucosal mRNA expressions of complement C3 and interleukin-17 in inflammatory bowel disease
โ Scribed by T. Sugihara; A. Kobori; H. Imaeda; T. Tsujikawa; K. Amagase; K. Takeuchi; Y. Fujiyama; A. Andoh
- Book ID
- 108701171
- Publisher
- John Wiley and Sons
- Year
- 2010
- Tongue
- English
- Weight
- 347 KB
- Volume
- 160
- Category
- Article
- ISSN
- 0009-9104
No coin nor oath required. For personal study only.
๐ SIMILAR VOLUMES
## Abstract Herpesviruses have been suggested as possible etiological agents of ulcerative colitis and Crohn's disease. Recently, increased numbers of EpsteinโBarr virus (EBV)โinfected cells have been detected in ulcerative colitis as compared to Crohn's disease. Interestingly, expression levels of
Background: Interleukin (IL)-17F, produced in IL-23R-expressing Th17 cells, is a novel member of the IL-17 cytokine family. Given the association of IL23R with inflammatory bowel disease (IBD), we characterized the role of IL-17F in IBD including its intestinal gene expression and the effect of the