✦ LIBER ✦

The effects of glucosamine hydrochloride on subchondral bone changes in an animal model of osteoarthritis

✍ Scribed by Susanne X. Wang; Sheila Laverty; Mircea Dumitriu; Anna Plaas; Marc D. Grynpas

Publisher: John Wiley and Sons
Year: 2007
Tongue: English
Weight: 539 KB
Volume: 56
Category: Article
ISSN: 0004-3591
DOI: 10.1002/art.22574

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Does oral glucosamine prevent subchondra

Does oral glucosamine prevent subchondral bone loss in an animal model of osteoarthritis? Comment on the article by Wang et al

✍ Santos Castañeda; Raquel Largo; Emilio Calvo; Gabriel Herrero-Beaumont 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 32 KB 👁 2 views

In the recent study by Wang et al (1), the authors concluded that after anterior cruciate ligament transection (ACLT), changes in subchondral bone begin early and simultaneously with cartilage degradation and that oral glucosamine hydrochloride has a protective effect (1). They based this conclusion

Early identification of radiographic ost

Early identification of radiographic osteoarthritis of the hip using an active shape model to quantify changes in bone morphometric features: Can hip shape tell us anything about the progression of osteoarthritis?

✍ Jennifer S. Gregory; Jan H. Waarsing; Judd Day; Huibert A. Pols; Max Reijman; Ha 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 265 KB 👁 3 views

The impact of anterior cruciate ligament

The impact of anterior cruciate ligament injury on lubricin metabolism and the effect of inhibiting tumor necrosis factor α on chondroprotection in an animal model

✍ K. A. Elsaid; J. T. Machan; K. Waller; B. C. Fleming; G. D. Jay 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 366 KB 👁 2 views

## Abstract ## Objective To examine the effects of anterior cruciate ligament transection (ACLT) in a rat model on lubricin metabolism and its relationship to markers of inflammation and cartilage damage, and to determine whether blocking the metabolic effects of tumor necrosis factor α (TNFα) by