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The effect of prolonged imipramine and electroconvulsive shock treatment on calcium/calmodulin-dependent protein kinase II in the hippocampus of rat brain

✍ Scribed by Andrzej Pilc; Piotr Branski; Agnieszka Palucha; Jaroslaw Aronowski


Book ID
117188238
Publisher
Elsevier Science
Year
1999
Tongue
English
Weight
216 KB
Volume
38
Category
Article
ISSN
0028-3908

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## Abstract Excessive release of glutamate during transient cerebral ischemia initiates a cascade of events that leads to the delayed and selective death of neurons located in the hippocampus. Activity of calcium calmodulin kinase II (CaM kinase), a protein kinase critical to neuronal functioning,