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The effect of different mutations in the LDL receptor on plasma lipid concentrations and the age of onset of coronary artery disease in patients with familial hypercholestrolaemia (FH)

โœ Scribed by V. Gudnason; R. Whittall; I.N.M. Day; A.K. Soular; S.N. McCarthy; L. Day; D. Patterson; M. Seed; G.R. Thompson; J.D. Betteridge; S.E. Humphries

Book ID
118325863
Publisher
Elsevier Science
Year
1995
Tongue
English
Weight
105 KB
Volume
115
Category
Article
ISSN
0021-9150

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โœ Giancarlo Ghiselli; Antonio Gaddi ๐Ÿ“‚ Article ๐Ÿ“… 1994 ๐Ÿ› Elsevier Science ๐ŸŒ English โš– 415 KB

Elevation of blood cholesterol, low-density lipoproteins (LDL) and apolipoprotein B (apoB) are hallmarks of familial hypercholesterolemia (FH), a genetic condition caused by the defective functioning of the cellular receptor for apoB-100 in LDL. ApoB-100 is also present in lipoprotein(a) (Lp(a)). In