## Abstract ## Objective Anti–heat shock protein 60 autoantibodies (anti‐Hsp60) are associated with cardiovascular disease and are known to affect endothelial cells in vitro, and we have recently shown that anti‐Hsp60 promote thrombosis in a murine model of arterial injury. Based on those findings
The capacity of Drosophila to heat harden associates with low rates of heat-shocked protein synthesis
✍ Scribed by Travis K. Johnson; Fiona E. Cockerell; Lauren B. Carrington; Lea Rako; Ary A. Hoffmann; Stephen W. McKechnie
- Publisher
- Elsevier Science
- Year
- 2009
- Tongue
- English
- Weight
- 167 KB
- Volume
- 34
- Category
- Article
- ISSN
- 0306-4565
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