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The Aβ1–42M35C mutated amyloid peptide Aβ1–42 and the 25–35 fragment fail to mimic the subtype-specificity of actions on recombinant human nicotinic acetylcholine receptors (α7, α4β2, α3β4)

✍ Scribed by Luanda J. Pym; Steven D. Buckingham; Victor Tsetlin; C.A.R. Boyd; David B. Sattelle


Book ID
116769848
Publisher
Elsevier Science
Year
2007
Tongue
English
Weight
467 KB
Volume
427
Category
Article
ISSN
0304-3940

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