## Abstract ## Background Duchenne muscular dystrophy (DMD) is an Xβlinked recessive muscle wasting disorder characterised by the absence of the protein dystrophin. Antisense oligonucleotides have been used to reβdirect dystrophin preβmRNA processing by blocking sequences crucial to preβmRNA splic
β¦ LIBER β¦
Terminal antisense oligonucleotide modifications can enhance induced exon skipping
β Scribed by Bijanka L. Gebski; Stephen J. Errington; Russell D. Johnsen; Susan Fletcher; Stephen D. Wilton
- Book ID
- 116792347
- Publisher
- Elsevier Science
- Year
- 2005
- Tongue
- English
- Weight
- 280 KB
- Volume
- 15
- Category
- Article
- ISSN
- 0960-8966
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