𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Teratogen-induced activation of ERK, JNK, and p38 MAP kinases in early postimplantation murine embryos

✍ Scribed by Mirkes, Philip E. ;Wilson, Kathleen L. ;Cornel, Leanne M.

Publisher
John Wiley and Sons
Year
2000
Tongue
English
Weight
472 KB
Volume
62
Category
Article
ISSN
0040-3709

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Inhibition of the p38 pathway upregulate
Inhibition of the p38 pathway upregulates macrophage JNK and ERK activities, and the ERK, JNK, and p38 MAP kinase pathways are reprogrammed during differentiation of the murine myeloid M1 cell line
✍ J. Perry Hall; Roger J. Davis 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 456 KB

Mitogen-activated protein (MAP) kinases have been implicated as important mediators of the inflammatory response. Here we report that c-Jun NH(2)-terminal kinase (JNK), extracellular signal-regulated kinase (ERK), and p38 MAP kinase activities are reprogrammed during the IL-6 induced macrophage-like

Reciprocal regulation of ATPγS-induced m
Reciprocal regulation of ATPγS-induced monocyte chemoattractant protein-1 production by ERK and p38 MAP kinases in rat corticostriatal slice cultures
✍ Takahiro Katayama; Misato Ito; Shuji Kaneko; Masamichi Satoh; Takashi Uehara; Ma 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 654 KB

## Abstract Monocyte chemoattractant protein‐1 (MCP‐1, CCL2) is a well‐defined chemokine implicated in the pathology of various neurodegenerative diseases and brain injuries, such as Alzheimer's disease, multiple sclerosis, stroke, and traumatic injury. We investigated the effect of the activation

Inhibition of p38MAP kinase potentiates
Inhibition of p38MAP kinase potentiates the JNK/SAPK pathway and AP-1 activity in monocytic but not in macrophage or granulocytic differentiation of HL60 cells
✍ Xuening Wang; George P. Studzinski 📂 Article 📅 2001 🏛 John Wiley and Sons 🌐 English ⚖ 240 KB

## Abstract Monocytic differentiation of HL60 cells induced by 1,25‐dihydroxyvitamin D~3~ (1,25 D~3~) has been recently shown (Exp Cell Res 258, 425, 2000) to be enhanced by an exposure to SB203580 or to SB202190, specific inhibitors of p38MAP kinase, with concomitant up‐regulation of the c‐jun N t

PMA-induced activation of the p42/44ERK-
PMA-induced activation of the p42/44ERK- and p38RK-MAP kinase cascades in HL-60 cells is PKC dependent but not essential for differentiation to the macrophage-like phenotype
✍ Heidi Schultz; Katrin Engel; Matthias Gaestel 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 201 KB 👁 1 views

The signaling mechanisms leading to phorbol ester myristate (PMA)-induced differentiation of HL-60 cells to the macrophagelike phenotype were investigated by using different protein kinase inhibitors. The protein kinase C inhibitor Ro 31-8220 specifically blocks PMA-induced differentiation, activati

Ultraviolet radiation (UVR) activates p3
Ultraviolet radiation (UVR) activates p38 MAP kinase and induces post-transcriptional stabilization of the C/EBPδ mRNA in G0 growth arrested mammary epithelial cells
✍ Bin Li; Junling Si; James W. DeWille 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 358 KB

## Abstract The G~0~ growth arrest (quiescent) state is highly conserved in evolution to promote survival under adverse environmental conditions. To maintain viability, G~0~ growth arrested cells limit gene expression to essential growth control and pro‐survival genes. CCAAT enhancer binding protei