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Subcellular distribution and autophosphorylation of calcium/calmodulin-dependent protein kinase II-α in rat hippocampus in a model of ischemic tolerance

✍ Scribed by M. Shamloo; F. Kamme; T. Wieloch


Book ID
117493342
Publisher
Elsevier Science
Year
2000
Tongue
English
Weight
440 KB
Volume
96
Category
Article
ISSN
0306-4522

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## Abstract Excessive release of glutamate during transient cerebral ischemia initiates a cascade of events that leads to the delayed and selective death of neurons located in the hippocampus. Activity of calcium calmodulin kinase II (CaM kinase), a protein kinase critical to neuronal functioning,