## Abstract Plasma membranes purified from RBL‐5 leukemia cells and solubilized with Na deoxycholate were fractionated on an Ultrogel AcA34 column. Fractions containing most of the tumor‐rejection activity but low amounts of gp70 and p30 were consolidated and used to hyperimmunize semisyngeneic, CB
Structure of the lipid phase of rauscher murine leukemia virus
✍ Scribed by Landsberger, Frank R. ;Lenard, John ;Compans, Richard W. ;Paxton, John
- Publisher
- Wiley (John Wiley & Sons)
- Year
- 1972
- Tongue
- English
- Weight
- 218 KB
- Volume
- 1
- Category
- Article
- ISSN
- 0091-7419
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract Rauscher murine leukemia virus (R‐MuLV) induces a rapidly developing erythroleukemia in BALB/c mice. Previously, we have shown that mouse interferon‐α/β (Mu IFN‐α/β) applied shortly after virus inoculation efficiently inhibits the leukemic process (Hekman __et al.__, 1981). Here we desc
## Abstract The expression of murine leukemia virus structural polypeptides on the surface of cells producing exogenous Friend leukemia virus, endogenous ecotropic AKR and xenotropic BALB/c virus was investigated. Antisera to Friend virus gp71, p30, p15E, p12 and p10 were employed in a complement‐d
## Abstract Antigens present in gp70 and p30 purified from Rauscher virus, were tested for immunogenicity in various assays measuring the anti‐tumor immune response against lymphocytic leukemias of Friend (FBL‐3) or Rauscher (RBL‐5) virus origin. p30 had no effect on __in vitro__ cytotoxicity again
## Abstract By an in vitro microcytotoxicity assay, thymocytes from mice infected with Moloney murine leukemia virus since birth (MuLV‐M carriers) caused a dramatic reduction of normal, non‐infected syngeneic target cells; they usually spared identically derived target cells infected with MuLV‐M. T