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Stimulation of vesicular monoamine transporter 2 activity by DJ-1 in SH-SY5Y cells

✍ Scribed by Shizuma Ishikawa; Yuki Tanaka; Kazuko Takahashi-Niki; Takeshi Niki; Hiroyoshi Ariga; Sanae M.M. Iguchi-Ariga

Book ID
116303382
Publisher
Elsevier Science
Year
2012
Tongue
English
Weight
389 KB
Volume
421
Category
Article
ISSN
0006-291X

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Mutant torsinA, which causes early-onset
Mutant torsinA, which causes early-onset primary torsion dystonia, is redistributed to membranous structures enriched in vesicular monoamine transporter in cultured human SH-SY5Y cells
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## Abstract A single GAG deletion in the __DYT1__ gene causes primary early‐onset, generalized torsion dystonia. The __DYT1__ protein product, torsinA, belongs to the AAA+ family of proteins. When overexpressed, wild‐type torsinA localizes mainly to the endoplasmic reticulum, whereas the mutant for