𝔖 Bobbio Scriptorium
✦   LIBER   ✦

STAT3 mediates the survival signal in oncogenic ras-transfected intestinal epithelial cells

✍ Scribed by Shinichiro Zushi; Yasuhisa Shinomura; Tatsuya Kiyohara; Yoshiji Miyazaki; Shinya Kondo; Masamitsu Sugimachi; Yoshifumi Higashimoto; Shuji Kanayama; Yuji Matsuzawa

Publisher
John Wiley and Sons
Year
1998
Tongue
French
Weight
99 KB
Volume
78
Category
Article
ISSN
0020-7136

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Oncogenic H-Ras and PI3K signaling can i
Oncogenic H-Ras and PI3K signaling can inhibit E-cadherin-dependent apoptosis and promote cell survival after photodynamic therapy in mouse keratinocytes
✍ Jesús Espada; Sergio Galaz; Francisco Sanz-Rodríguez; Alfonso Blázquez-Castro; J 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 345 KB

## Abstract Maintenance of E‐cadherin mediated cell–cell contacts is often required for the survival of epithelial cells and tissues. Here we report that oncogenic activation of H‐Ras in murine keratinocytes can prevent cell death induced by immunological disruption of E‐cadherin adhesion. A simila

Fak/Src signaling in human intestinal ep
Fak/Src signaling in human intestinal epithelial cell survival and anoikis: Differentiation state-specific uncoupling with the PI3-K/Akt-1 and MEK/Erk pathways
✍ Véronique Bouchard; Marie-Josée Demers; Sonya Thibodeau; Vincent Laquerre; Naoya 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 429 KB 👁 2 views

## Abstract Human intestinal epithelial cell survival and anoikis are distinctively regulated according to the state of differentiation. In the present study, we analyzed the roles of focal adhesion kinase (Fak)/Src signaling to the PI3‐K/Akt‐1 and mitogen‐activated protein kinase (MEK)/extracellul

The cannabinoid δ9-tetrahydrocannabinol
The cannabinoid δ9-tetrahydrocannabinol inhibits RAS-MAPK and PI3K-AKT survival signalling and induces BAD-mediated apoptosis in colorectal cancer cells
✍ Alexander Greenhough; Helena A. Patsos; Ann C. Williams; Christos Paraskeva 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 French ⚖ 310 KB

## Abstract Deregulation of cell survival pathways and resistance to apoptosis are widely accepted to be fundamental aspects of tumorigenesis. As in many tumours, the aberrant growth and survival of colorectal tumour cells is dependent upon a small number of highly activated signalling pathways, th

Oncostatin M–induced CCL2 transcription
Oncostatin M–induced CCL2 transcription in osteoblastic cells is mediated by multiple levels of STAT-1 and STAT-3 signaling: An implication for the pathogenesis of arthritis
✍ Sang-Heng Kok; Chi-Yuan Hong; Mark Yen-Ping Kuo; Chih-Chiang Wang; Kuo-Liang Hou 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 393 KB 👁 2 views

## Abstract ## Objective To examine the roles of STATs 1 and 3 in CCL2 production in human osteoblastic cells and their influences on arthritis development. ## Methods The expression of CCL2 in primary human osteoblasts and U2OS human osteoblastic cells was examined by Northern blotting and enzy

1α,25-dihydroxyvitamin D3-mediated stimu
1α,25-dihydroxyvitamin D3-mediated stimulation of steroid sulphatase activity in myeloid leukaemic cell lines requires VDRnuc-mediated activation of the RAS/RAF/ERK-MAP kinase signalling pathway
✍ Philip J. Hughes; Geoffrey Brown 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 448 KB

## Abstract 1α,25‐dihydroxyvitamin D~3~ (1α,25(OH)~2~D~3~) stimulates the activity of steroid sulphatase (STS) in myeloid cells [Hughes et al., 2001, 2005]. This was attenuated by inhibitors of phospholipase D (PLD) (__n__‐butanol, 2,3‐diphosphoglyceric acid, C~2~‐ceramide) and phosphatidate phosph