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Soluble phosphorylated fms-like tyrosine kinase: III. FLT3 protein in patients with acute myeloid leukemia (AML)

✍ Scribed by F. Ravandi; I. Jilani; E. Estey; H. Kantarjian; A. Dey; C. Aguilar; C. Jitkaroon; F. Giles; S. O’Brien; M. Keating; M. Albitar

Book ID: 104040310
Publisher: Elsevier Science
Year: 2007
Tongue: English
Weight: 499 KB
Volume: 31
Category: Article
ISSN: 0145-2126
DOI: 10.1016/j.leukres.2006.11.001

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## Abstract ## BACKGROUND The clinical relevance of mutations of the __FMS__‐like tyrosine kinase 3 (__FLT3__) gene in specific cytogenetic subgroups is not clear. The authors examined internal tandem duplication (ITD) and Asp835 mutations of __FLT3__ in patients with acute promyelocytic leukemia